IndraLab

Statements

Lipopolysaccharide bound to TLR4 inhibits MYD88. 2 / 2
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"Convincing researches have indicated that LPS, a major component of Gram negative bacterial cytoderm, binds to TLR4 and triggers MyD88 mediated signaling cascades activation of NF-kappaB, and eventually leads to inflammatory response."
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"Moreover, the competitive binding of gCTRP9 and LPS to the myeloid differentiation protein 2 (MD2) and TLR4 complex was associated with direct binding to MD2, thereby inhibiting the downstream signaling molecule MyD88."
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