IndraLab

Statements

Reactive Oxygen Species inhibits STAMBPL1. 2 / 2
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"Thus, H. pylori infection causes excessive OS and produces redundant ROS, which in turn downregulates the STAMBPL1 and contributes to the survival of GBC cells."
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"Recent studies have shown that excessive ROS generated in H. pylori infection induces direct degradation of STAMBPL1 by cullin 1-RING ubiquitin ligase and the 26S proteasome, while STAMBPL1 can lead to cell apoptosis by reducing the anti-apoptotic protein survivin (Chaithongyot and Naumann, 2022)."
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