IndraLab

Statements


USP39 deubiquitinates ZEB1. 2 / 2
| 2

reach
"Collectively, our findings suggest that the balanced deubiquitination and ubiquitination of ZEB1 by USP39 and TRIM26 represent a novel mechanism for the progression of HCC, and this discovery provides a promising strategy for targeting USP39 or TRIM26 in the treatment of HCC cases with aberrant ZEB1 expression levels."

reach
"USP39 promotes EMT and inhibits ZEB1 ubiquitination in HCC."