IndraLab

Statements



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"In addition, OTUB1 enhances glycolysis and epithelial-mesenchymal transition of endometriosis cells, leading to promote proliferation, migration, and invasion of endometriosis cells."

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"Further, we found that OTUB1 mediated glycolysis by regulating TGFBI in infantile hemangioma."

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"OTUB1 promotes the progression of acute myeloid leukemia by regulating glycolysis via deubiquitinating c-Myc."

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"Loss of OTUB1 in H1299 cells led to decreased compensatory glycolysis significantly compared to wildtype H1299 cells under hypoxia (Fig. 7K, L)."

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"In conclusion, OTUB1 promotes glycolysis in endometriosis cells."

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"And OTUB1 promoted glycolysis in endometriosis cells ."

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"Further , we found that OTUB1 mediated glycolysis by regulating TGFBI in infantile hemangioma ."

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"Mechanistically, OTUB1 drives the malignant development of AML through regulating cellular aerobic glycolysis by deubiquitinating c-Myc."

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"In brief, our results revealed a novel mechanism by which OTUB1 promotes glycolysis via deubiquitinating c-Myc in AML."

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"And OTUB1 promoted glycolysis in endometriosis cells."

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"The results showed that OTUB1 promoted glycolysis via HSF1-mediated PFKFB3 expression ( Figures S4A and S4B ) ."