IndraLab

Statements

USP49 inhibits apoptotic process. 6 / 6
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"Besides, USP49 inhibits I/R‐induced cell viability suppression and apoptosis in AC16 cells via the DUSP1‐JNK1/2 pathways (Zhang et al., 2019)."
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"USP49 facilitated the deubiquitination of Axin and suppressed IL-1β-stimulated apoptosis of chondrocytes by inhibition of Wnt/β-catenin signaling [7]."
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"This demonstrated that USP49 knockdown significantly increased cell apoptosis, while GRβ introduction overcame this effect."
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"USP49 inhibits ischemia-reperfusion-induced cell viability suppression and apoptosis in human AC16 cardiomyocytes through DUSP1-JNK1/2 signaling."
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"Furthermore, USP49 overexpression in Y‐79 cells increased cell proliferation and inhibited cell apoptosis, whereas the knockdown of USP49 in Y‐79/CBP cells exerted an opposite effect (Figure 2F–H)."
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"Overexpression of USP49 promoted cell proliferation, inhibited cell apoptosis, and increased the IC50 value of CBP in RB cells."
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