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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

KCNMA1 activates SULT1A1. 2 / 2

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"This analysis thus further confirms that abnormally elevated neurotransmitter release and STP caused by the absence of FMRP are mediated by BK channel dependent, excessive AP broadening that occurs in the axons in the vicinity of presynaptic terminals."

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"If this is the case, then BK channel blockers should reduce or abolish the differences in synaptic transmission and STP between WT and Fmr1 KO mice."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...