IndraLab

Statements


SLC2A1 binds NRP1 and env. 3 / 3
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sparser
"These results clearly indicated that specific infection was successfully induced through the interaction between HTLV-1 Env protein at the cell surface and receptor molecule(s) incorporated into the VSV particles—particularly the functional association of the Env with GLUT1 or NRP1 proteins."

sparser
"It was also observed that GLUT1 and NRP-1 could form intracytoplasmic complexes in transfected cells, an association that was greatly enhanced in the presence of Env [ xref ]."

sparser
"The fact that NRP-1 and GLUT1 can form a complex in the presence of HTLV-1 Env suggested that these two molecules work together to promote HTLV-1 entry."