IndraLab

Statements


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"WB and CoIP analyses indicated that the TNF-DAG1 axis mainly mediated the MES state of MES-like cancer cells, and the TNF-CELSR2 axis-mediated MES state was predominantly in NPC-like cancer cells ( xref H–J)."

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"We also demonstrate that TNF interacts with CELSR2, activates the downstream p65 signaling and concurrently elevates the lactylation level of HDAC1."

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"Thus, we focused on TNF-CELSR2 mediates the conversion from NPC-like to MES-like GBM cells."

No evidence text available