IndraLab

Statements


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"BTK inhibitor PCI-32765 promoted CYLD dependent apoptosis in rituximab resistant non-GCB-DLBCL cells."

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"Previous researches in CLL indicated that BTK inhibitor ibrutinib could largely increase CYLD activity through increasing CYLD miRNA transcription, which could inhibit cells proliferation in CLL [XREF_BIBR]."