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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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PSMD14 activates Cell Survival. 4 / 4

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"PSMD14 siRNA knockdown in another human cell line, hTERT immortalized human mammary epithelial cell line hTERT-HMEC1, caused a dramatic reduction of cell viability (36% viability)."

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"Conversely, ectopic POH1 expression substantially potentiated tumour cell survival in detached culture conditions (XREF_FIG)."

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"To more specifically test for any requirement of the zinc metalloproteinase motif of Poh1 to support cell viability and proteasome function, we developed a RNAi complementation strategy."

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"We studied the oncogenic function of POH1 in solid tumors; knockdown of POH1 expression in QGY-7701, EC109, and HCT116 cells decreased cell viability and induced apoptosis."

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PSMD14 activates Cell Survival. 4 / 4

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