IndraLab

Statements

PSMD14 activates Cell Survival. 10 / 10
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"The results showed that MYSM1 or EIF3H overexpression decreased cell viability, while PSMD14 overexpression increased cell viability under DOX treatment (Fig. 1C)."
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"To more specifically test for any requirement of the zinc metalloproteinase motif of Poh1 to support cell viability and proteasome function, we developed a RNAi complementation strategy."
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"Conversely, ectopic POH1 expression substantially potentiated tumour cell survival in detached culture conditions (XREF_FIG)."
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"The results suggested that PSMD14 overexpression increased the cell viability compared with the negative control over time, while PTBP1 knockdown reversed the effect ( Fig. 5 c)."
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"POH1 is essential for viability, and POH1 knockdown inhibits proteasome activity and reduces cell survival [33]."
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"Indeed, overexpression of PSMD14 promoted proliferation and cell viability together with other pro-tumoral properties such as migration and invasion [ 39 , 49 , 52 ]."
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"PSMD14 knockdown inhibited PC cell viability, proliferation, migration, and invasion."
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"We studied the oncogenic function of POH1 in solid tumors; knockdown of POH1 expression in QGY-7701, EC109, and HCT116 cells decreased cell viability and induced apoptosis."
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"PSMD14 siRNA knockdown in another human cell line, hTERT immortalized human mammary epithelial cell line hTERT-HMEC1, caused a dramatic reduction of cell viability (36% viability)."
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"CCK-8 assays demonstrated that the knockdown of PSMD14 significantly reduced osteosarcoma cell viability to around 50% of control shRNA-treated cells at 96 h (Fig. 2J, K)."
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