IndraLab

Statements

USP22 activates inflammatory response. 8 / 8
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"Silencing of USP22 suppressed ROS production and inflammation while inhibition of USP14 reduced the accumulation of oxidized proteins."
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"In addition, USP22 expression increased the inflammatory response."
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"Accordingly , USP22 downregulation also attenuated cerebral I / R-induced oxidative stress , inflammation , and cell apoptosis in mice , thereby reducing nerve injury and neurological dysfunction [ 20 ] ."
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"Overexpression of USP22 ameliorates LPS-induced endometrial stromal cells inflammation and modulates cells decidualization by inhibiting ferroptosis."
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"USP22 contributes to the inflammatory response and apoptosis in podocytes through oxidative stress and secretion of inflammatory mediators, up-regulation of caspase-3, and an increase in the BAX/Bcl-2 ratio96."
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"Therefore, USP22/SIRT1 signaling pathway may contribute to liver inflammation in ALD."
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"Taken together, the findings of the present study have demonstrated for the first time that USP22 inhibition attenuates high glucose induced podocyte injuries and inflammation."
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"In the current study, we show that the overexpression of USP22 induced by treatment with FO greatly improved Ang II-induced cardiac dysfunction, heart hypertrophy, inflammation, and fibrosis (Figures 1–4)."
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