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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

TP53BP1 inhibits ATXN3. 1 / 1

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"Moreover, we found that replacing endogenous RNF8 with a catalytically inactive mutant (C403S; denoted as * RING), which impairs RNF8 dependent ubiquitylation and 53BP1 accumulation at DSBs (Mailand etal, 2007), did not impair accrual of ataxin-3 (Fig XREF_FIG F and G)."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...