IndraLab

Statements



reach
"Our results indicate that Cacna1c downregulation stimulates mitochondrial biogenesis under oxidative stress without affecting mitophagic processes, thereby promoting the turnover of mitochondria and preventing the accumulation of dysfunctional mitochondria in HT22 cells."

reach
"In the employed paradigm of oxidative glutamate toxicity, Cacna1c depletion also protected against detrimental mitochondrial fission and stimulated mitochondrial biogenesis without affecting mitophagy, thus promoting the turnover of mitochondria and preventing the accumulation of dysfunctional mitochondria in neuronal HT22 cells."