IndraLab

Statements

NOTCH1 binds TNF. 10 / 10
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"We also observed potential interacting pairs between EP_C1_LMP1 cells with activated Notch pathway and multiple cell types through NOTCH1-TNF and NOTCH2-JAG2, which have been related to radiation sensitivity xref and cancer stem-like side population cells xref in NPC (Supplementary Fig.  xref )."
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"JAM2–JAM3, C5AR1–RPS19, TNF–DAG1 and TNF–NOTCH1 signaling pathways are associated with OL maturation and myelination."
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"We observed that, some classical signaling such as TNF-NOTCH1 pair was expressed in all eight gained ImC-LSC, as reported, Notch and TNF signaling had all been verified to regulate the homeostasis of the corneal epithelium and the corneal inflammatory response and wound healing after injury [ xref – xref ]."
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"For the receptor-ligand interaction, Agrafioti et al. [ xref ] found that PVR-TIGIT, PVR-CD96, and PV-CD226 receptor/ligand pairs interact between M1 and T cells, while TNF-NOTCH1 and PVR-TNFSF9 receptor/ligand pairs interact between M1 and B cells, and many other cross-talks were enhanced in periodontitis-affected sites."
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"TNF promoted p52/RELB binding to NICD, which enhanced binding at the RBPjκ site within the Hes1 promoter."
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"By linking Notch and TNFalpha it was shown that (1) Notch-1 mucosal expression differs in inflamed and non inflamed mucosa and increases in response to anti-TNFalpha treatment; (2) Notch-1 function is regulated by TNFalpha inhibitors; (3) Notch-1 binds to TNFalpha; and (4) Notch-1 inhibition prevents anti-TNFalpha-induced T cell cycle arrest but not apoptosis."
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"By linking Notch and TNFα it was shown that (1) Notch-1 mucosal expression differs in inflamed and non-inflamed mucosa and increases in response to anti-TNFα treatment; (2) Notch-1 function is regulated by TNFα inhibitors; (3) Notch-1 binds to TNFα; and (4) Notch-1 inhibition prevents anti-TNFα-induced T cell cycle arrest but not apoptosis."
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"In addition, the interaction of NOTCH1-TNF, TGFB1-TGFβ receptor and CD47-SIRPA were also found between malignant T cells and LAMP3 + cDCs ( Fig. 5 F)."
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"The upregulation of constitutive nuclear factor NF-κB, as well as CD30, CD40, tumor necrosis factor (TNF)-α, and Notch1 interactions contributes to these processes ( xref )."
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"In parallel with our findings, Daniel et al. [43] reported that TNF-α inhibited Mfn2 expression in cells and that Notch1 could directly bind to TNF-α [44] , supporting positive regulation of Mfn2[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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