IndraLab

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CACNA1A activates apoptotic process. 23 / 23
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"Mhp Can Induce Apoptosis to PAMs by Nitric Oxide and (ROS) Production."
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"Thereafter, Mhp can adhere to the cilium surface and spread through the respiratory tract, destroying cilium structures and causing apoptosis to epithelial cells (60)."
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"Partial pattern diagram of Mhp infection is shown in Figure 2, Mhp infection destroys cilium structures and induces the apoptosis of epithelial cells, which may increase the risk of secondary infections."
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"Whether early or late apoptosis, the proportion of cells induced by MHP was similar to that of radiation (5 Gy), while being greater than that of MH."
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"However, Mhp-induced apoptosis of alveolar macrophages prevents this process from proceeding properly."
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"For example, VP51, a tumor necrosis factor receptor (TNFR) like homolog, enhanced virus replication via affecting cell proliferation and inhibiting apoptosis induced by SGIV in Fathead minnow (FHM) ce[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Follow-up studies reported Mhp infection could induce apoptosis to PBMCs by ROS and NO production, which were related to the Bax/Bcl-2, p38 MAPK signaling pathway and caspase activation (93)."
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"Mhp infection decreases host immunity and induces significant apoptosis or dysfunction to immune cells, as well as increasing the secondary viral or bacterial infection risk.With the development of modern swine production goes to larger-scale and intensive, coinfections of Mhp and other pathogens are becoming common and serious."
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"We presume that BI-TK and GCV treatment system is able to prevent tumor growth and induce apoptosis in the rodent bladder cancer model by down-regulating Prx-I expression."
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"It is known that SGIV infection of FHM induces canonical apoptosis and facilitates the activation of caspase 3/8/9, which is associated with the activation of p53 and AP-1 pathways [48,49,50]."
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"The BI-TK and GCV treatment system exhibited a sustainable anti-tumor growth activity and induced apoptosis in bladder cancer tissues by inhibition of Prx-I through the NF-kappaB pathway."
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"We overexpressed Ec-CD in FHM cells, analyzed Ec-CD influence on SGIV-induced apoptosis by flow cytometry and detected caspase activity."
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"Mhp can also induce immune cells apoptosis by ROS production, excessive NO release and caspase activation, and increases the risk of coinfection with other pathogens."
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"The BI-TK and GCV system (group C) was shown to inhibit tumor growth and caused the apoptosis of tumor cells as assessed by in situ terminal transferase dUTP nick-end labeling assay."
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"Specific association of SCA6 pathology clearly implies that small CAG expansion of the CACNL1A4 gene disturbs normal functions of the alpha 1A Ca 2+ channel, and ultimately produce apoptosis of Purkin[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"In addition, TRAF6 can inhibit the apoptosis induced by SGIV in FHM cells."
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"It is conceivable that the initial MHP of SLE lymphocytes would trigger necrosis rather than apoptosis if treated with exogenous H 2 O 2 , resulting in the exaggerated inflammation cascade seen in SLE[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"The late-stage apoptosis induced by the Mhp strain JS or 168L in hTERT-PTECs was significantly decreased when rNFOR was blocked with an anti-rNFOR antibody."
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"The late-stage apoptosis induced by either the high-virulence Mhp strain JS or the low-virulence Mhp strain 168L in hTERT-PBECs was significantly decreased when rNFOR was blocked with an anti-rNFOR polyclonal antibody, with late-stage apoptotic cell percentages decreasing by 41.9 and 35.8%, respectively."
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"Evidently, SGIV-VP96 overexpression had a preventive effect on apoptosis induced by SGIV infection in FHM cells.Apoptosis is an important tactic for host to limit viral replication by killing virus in[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"Therefore, persistent MHP, causing increased ROI production (a trigger of apoptosis) and depletion of ATP (required for AICD), might be responsible for the paradox of increased spontaneous apoptosis a[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"FCM analyses of annexin-V, APCA, and PI revealed that lentivirus-mediated inhibition of CYLD significantly prevented early and total apoptosis (LR and TR, respectively) but not late apoptosis (UR) when compared with the sh-GFP groups (Fig. 5B; LR 4.98 ± 0.94 vs. 7.15 ± 0.10, P = 0.016; UR 1.65 ± 1.14 vs. 3.51 ± 1.19, P = 0.122; TR 6.63 ± 0.21 vs. 10.67 ± 1.28, P = 0.029)."
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"Data were analyzed with the Student's t-test to compare means between GS/pcDNA3.1 and GS/pcDNA3.1-EcFADD cells at single time points.Typical apoptosis is induced by SGIV infection in non-host FHM cell[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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