IndraLab

Statements


CUL1 activates ATXN3. 1 / 1
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reach
"Knockdown of CUL1 abolished FBXL3 mediated reduction of normal ATXN3, but only accounted for about half of the observed FBXL3 facilitated decrease of pathogenic ATXN3 (XREF_FIG), suggesting that FBXL3 handles or recognizes normal and mutant ATXN3 differently."