IndraLab

Statements

USP17L2 activates cell population proliferation. 8 / 8
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"DUB3 promoted OSCC cells proliferation, while suppressing apoptosis via facilitating EZH2 production."
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"The knockdown of USP17 also suppressed cell proliferation and glycolysis ."
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"In summary, we found that DUB3 enhanced OSCC cells proliferation and xenograft tumor growth, while inhibited their apoptosis via promoting BRD4 mediated upregulation of EZH2."
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"Similar to our observations, previous shRNA studies have also indicated that Dub3 knockdown reduces proliferation by inhibiting the G1-S phase cell cycle progression and inhibits chemotaxis [14,15,26][MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"In vitro, Dub3 knockdown could inhibit the proliferation of ovarian cancer cells and increase cell apoptosis."
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"McFarlane et al. identify that USP17 is highly expressed in several tumor biopsies , and USP17 depletion significantly impairs G1-S phase transition and blocks cell proliferation ( 56 ) ."
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"Functionally, we found that the effect of DUB3 on cyclin A mediates proliferation of NSCLC cells."
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"DUB3 may also promote the proliferation of NSCLC by inhibiting Cyclin A degradation."
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