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OTUD5 leads to the dephosphorylation of TRIM28. 1 / 1

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"This shows that the residual ATM activity in OTUD5-deficient cells is inadequate to support sufficient phosphorylation of KAP1 , a key step in DNA repair in KAP1-rich genomic environments such as the telomeres and heterochromatin, thereby translating into a repair defect specifically in these regions."

39420004

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IndraLab

Statements

OTUD5 leads to the dephosphorylation of TRIM28. 1 / 1

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