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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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Statements

BAP1 increases the amount of OGT. 3 / 4

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"BAP1 also increases OGT protein levels, suggesting that BAP1 is a potential deubiquitinase for OGT as well (XREF_SUPPLEMENTARY)."

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"OGT and HCF-1 levels were decreased by Bap1 deletion, indicating a critical role for BAP1 in stabilizing these epigenetic regulators."

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"OGT and HCF-1 levels were decreased by Bap1 deletion, and were associated with myeloid transformation ."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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Statements

BAP1 increases the amount of OGT. 3 / 4

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