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USP4-C311A leads to the dephosphorylation of p38. 2 / 2

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"USP4 (C311A) mutation did not inhibit the TAK1 polyubiquitination, and phosphorylation of TAK1, = JNK1/2, and p38 in angiotensin treated myocytes."

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"In the same context, the overexpression of mutant USP4 C311A decreased the phosphorylation of AKT and p38 (Supplementary Figs."

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IndraLab

Statements

USP4-C311A leads to the dephosphorylation of p38. 2 / 2

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