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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP17L2 activates cell migration. 4 / 4

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"Moreover , Matrigel-Transwell analysis showed that suppression of USP17 decreased cell migration and invasion capacity ."

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"Dub3 is overexpressed in breast cancer; knockdown of Dub3 resulted in Snail1 destabilization, suppressed EMT and decreased tumour cell migration, invasion, and metastasis."

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"Dub3 expression markedly increased the cell migration and invasive capacity (XREF_FIG, XREF_SUPPLEMENTARY)."

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"Individual cell tracking also revealed Dub3 knockdown reduced the velocity and directionality of cell migration, and strongly inhibited the net distance of cell migration in MDA-MB231 and MDA-MB157 cells (XREF_FIG, XREF_SUPPLEMENTARY)."

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USP17L2 activates cell migration. 4 / 4

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