IndraLab

Statements


UCHL1 deubiquitinates SNCA. 2 / 2
1 | 1

reach
"No clear substrates have been described for UCHL1 in vivo, but UCHL1 may deubiquitinate alpha-synuclein."

"The deubiquitination of alpha-synuclein aggregates by USP9X favor their degradation by autophagy, while <span class="match term0">UCH-L1</span> contribute to the accumulation of alpha-synuclein by downregulating its lysosomal degradation."