IndraLab

Statements

KCNQ1 inhibits Kr. 2 / 2
| 2
reach
"Pharmacological inhibition and congenital defects (mutations in KCNQ1 , KCNH2 or KCNE family) of I Kr or I Ks exaggerate this innate repolarization disparity facilitating arrhythmia under specific con[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
22841828
reach
"Taken together, KCNQ1 (including KCNQ1 R174C mutant channels) attenuated I Kr when co-expressed with hERG."
29449639