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Statements

USP22 activates Fibrosis. 2 / 2
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"In the current study, we show that the overexpression of USP22 induced by treatment with FO greatly improved Ang II-induced cardiac dysfunction, heart hypertrophy, inflammation, and fibrosis (Figures 1–4)."
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"OTUD1 and USP22 have been shown to promote fibrosis and injury in renal tubular epithelial cells ."
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