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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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FOXO4 increases the amount of USP10. 3 / 3

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"In addition, FoxO4 modulated USP10 transcription."

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"This study aimed to investigate whether forkhead box O4 (FoxO4) could negatively modulate ubiquitin specific peptidase 10 (USP10) transcription to aggravate the apoptosis and oxidative stress of hypoxia and reoxygenation (H/R)-induced cardiomyocytes through Hippo and YAP pathway."

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"In conclusion, FoxO4 negatively modulated USP10 transcription to aggravate the apoptosis and oxidative stress of H/R induced H9C2 cells via blocking Hippo and YAP pathway."

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FOXO4 increases the amount of USP10. 3 / 3

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