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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

OTUB1 inhibits MAF. 3 / 3

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"As shown in Fig. 2c , Otub1 prevented c-Maf from polyubiquitination , while Nam increased c-Maf ubiquitination and partly rescued the ubiquitination level in the presence of Otub1 ( Fig. 2c ) , suggesting that Nam antagonized Otub1 in terms of c-Maf polyubiquitination ."

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"Otub1 abrogates c-Maf K48 linked polyubiquitination thus preventing its degradation and enhancing its transcriptional activity."

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"In other words, Otub1 is essential for AVT to inhibit c-Maf activity."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...