IndraLab

Statements

KCNQ1 activates HMR-1556. 2 / 2
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reach
"Wild-type (WT) KCNQ1 or the familial AF mutation KCNQ1 S140G were heterologously co-expressed with KCNE1 to enable electrophysiological recording of the slow delayed rectifier current (I Ks) and investigation of pharmacological effects of the I Ks selective blocker HMR-1556."
24006450
reach
"Wild-type (WT) KCNQ1 or the familial atrial fibrillation mutation KCNQ1 S140G was heterologously coexpressed with KCNE1 to enable electrophysiological recording of the slow delayed rectifier current (IKs) and investigation of pharmacological effects of the IKs selective blocker HMR-1556."
24006450