IndraLab

Statements


KCNQ1 activates HMR-1556. 1 / 1
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reach
"Wild-type (WT) KCNQ1 or the familial AF mutation KCNQ1 S140G were heterologously co-expressed with KCNE1 to enable electrophysiological recording of the slow delayed rectifier current (I Ks) and investigation of pharmacological effects of the I Ks selective blocker HMR-1556."