IndraLab

Statements



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"32 OTUD7B and MINDY1 could promote breast cancer proliferation by stabilizing ERα in a deubiquitylation activity–dependent manner."

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"These results indicated that MINDY1 promoted bladder cancer cell proliferation, at least partially, via the regulation of YAP."

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"MINDY1 promotes breast cancer cell proliferation by stabilizing estrogen receptor alpha."

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"Our results demonstrated that depletion of MINDY1 significantly decreased cell proliferation and increased the population in G1 phases , indicating that MINDY1 may regulate G1 to S transition in bladder cancer cells ( Fig. 5A , B ) ."

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"MINDY1 depletion significantly decreased bladder cancer cell proliferation."

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"We also found that MINDY1 promoted cell proliferation through YAP ."

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"Further investigations revealed that MINDY1 promoted the proliferation and migration of breast cancer cells through ERα."

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"Our data further demonstrated that MINDY1 depletion dramatically decreased the proliferation bladder cancer cells."

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"These results demonstrated that MINDY1 promoted bladder cancer proliferation through increasing YAP stability."

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"It was found that MINDY1 depletion inhibited cell proliferation in both vehicle and estradiol treated conditions (Fig. 7A and Fig. S3A)."

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"Correction to: MINDY1 promotes breast cancer cell proliferation by stabilizing estrogen receptor α."

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"We also found that MINDY1 promoted cell proliferation through YAP."

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"Our results demonstrated that depletion of MINDY1 significantly decreased cell proliferation and increased the population in G1 phases, indicating that MINDY1 may regulate G1 to S transition in bladder cancer cells (Fig. 5A, B)."

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"Consistently, silencing of MINDY1 reduced proliferation of bladder cancer cells."

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"Overexpression of YAP abrogated the MINDY1 depletion-induced inhibition of cell proliferation in bladder cancer cells (88)."

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"In addition, MINDY1 could promote the proliferation and migration of breast cancer cells by stabilizing ERα."