IndraLab

Statements

SLC16A1 activates USP38. 3 / 3
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"MCT1 knockdown impaired the ability of USP38 to reduce PM2.5‐induced lactate accumulation (Figure 6A) and abolished the protective effect of USP38 on cell viability (Figure 6B)."
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"Western blot analysis confirmed that MCT1 knockdown completely eliminated the ability of USP38 to inhibit PM2.5‐induced apoptosis (Figure 6C)."
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"TUNEL staining provides additional evidence that the suppression of MCT1 markedly diminishes the protective role of USP38 in counteracting PM2.5‐induced apoptotic cell death (Figure 6D,E)."
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