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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP27X activates SETD3. 4 / 4

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"Knockdown of USP27 by short hairpin RNA (shRNA) accelerated the degradation of SETD3 and blocked cell proliferation."

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"Since upregulation of SETD3 can promote liver tumorigenesis and cancer progression [12], it is possible that USP27 can also enhance metastatic phenotype by stabilizing SETD3."

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"Inhibition of USP27 expression impairs the pro-migratory ability of SETD3."

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"In conclusion, our data suggested that USP27 could positively regulate SETD3 protein stability."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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USP27X activates SETD3. 4 / 4

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