IndraLab

Statements

USP49 activates apoptotic process. 4 / 4
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"Inhibiting JNK1/2 activation significantly inhibited USP49 knockdown induced the cell viability inhibition, apoptosis and the JNK1/2 activation in AC16 cardiomyocytes."
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"On the contrary, USP49 knockdown suppressed cell proliferation, increased cell apoptosis, and reduced the IC50 value of CBP in CBP‐resistant RB cells."
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"USP49 knockdown Inhibits JNK1/2 activation and cell apoptosis [192]."
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"In consistent with this notion, USP49 knockdown was recently reported to result in a decrease of cell proliferation and invasion as well as an increase of cell apoptosis in glioblastoma cells [ 25 ]."
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