IndraLab

Statements

EIF3H activates apoptotic process. 9 / 9
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"Knockdown of EIF3H promotes apoptosis in HCT116 and SW1116 cells."
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"Knockdown of EIF3H led to cell cycle arrest and promoted apoptosis in OS cells."
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"These results indicated that suppression of EIF3H had a significant negative effect on colon cancer cell growth.Knockdown of EIF3H promoted apoptosis in human CRC cellsAs shown in Figure 4 (a), the percentages of early apoptotic cells (Annexin V+ and 7-AAD-) and late apoptotic cells (Annexin V+ and 7-AAD+) were significantly higher in shEIF3H infected HCT116 cells and obviously higher in shEIF3H infected SW1116 cells when compared to the corresponding numbers in control cells."
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"Taken together, these results confirmed that EIF3H accelerated the proliferation and migration through increasing OGT stability.Ferroptosis is a new non-apoptotic programmed cell death triggered by lipid ROS and has been regarded as a potential therapeutic target for cancer therapy."
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"The inhibition of CYPOR increases drug accumulation, and the EIF3H/HIF1α axis induces DNA break-mediated apoptosis, resulting in enhanced activity of DOX in tongue cancer cells."
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"Knockdown of EIF3H inhibits the development and progression of pancreatic cancer by regulating cell proliferation and apoptosis in vitro."
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"Taken together, these results suggested that the lentivirus expressing shEIF3H may significantly downregulate the expression of EIF3H in MM cells.Knockdown of EIF3H inhibited cell proliferation and in[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"In addition, we showed that EIF3H knock-down led to cell apoptosis."
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"Furthermore, the loss-of-function assays demonstrated that knockdown of EIF3H could inhibit the progression of pancreatic cancer cells by reducing proliferation capacity, promoting apoptosis, arresting cell cycle in G2 and suppressing cell migration."
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