IndraLab

Statements

USP27X activates BCL2L11. 3 / 3
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"Cathepsin K inhibition-induced mitochondrial ROS enhances sensitivity of cancer cells to anti-cancer drugs through USP27x-mediated Bim protein stabilization."
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"Our previous study suggested that cathepsin K inhibition enhances anti-cancer drug-induced apoptotic cell death through the upregulation of USP27x-mediated Bim pro-apoptotic protein [12]."
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"USP27x had no affect the phosphorylation of Bim, but increases stability of Bim by inhibition of ubiquitination [41]."
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