IndraLab

Statements

USP3 deubiquitinates KLF5. 4 / 5
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"These findings imply that the UCHL1-KLF5 axis might be a valuable therapeutic target for TNBC.Besides UCHL1, some other DUBs, such as ATXN3L, BAP1, and USP3 can deubiquitinate KLF5 and are implicated in the pathological development of breast cancer [29, 41, 42]."
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"For example, ATXN3L and USP3 mediate the deubiquitination of KLF5 and thus regulate the proliferation of BC cells [39,40]."
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"USP3 promotes breast cancer cell proliferation by deubiquitinating KLF5."
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"For example, USP3 regulates cancer progression and metastasis by deubiquitinating the Kruppel-like factor 5 (KLF5), stabilizing p53 or deubiquitination-dependent COL9A3/COL6A5."
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