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Infections activates TNFAIP3. 7 / 7
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"This agrees with Porcella et al. whose team found that plasmid-bearing C. trachomatis infection upregulated TNFAIP3 more significantly compared to its plasmidless counterpart."
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"Another similar study showed that cp BVDV infection significantly upregulated two negative NF-κB regulators, IER3 and TNFAIP3, thus possibly blocking this signaling pathway (Villalba et al., 2016)."
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"As expected, infection triggered an important upregulation of the CXCL2, CXCL8, CCL20, TNFAIP3, NFKBIA, and NFκBIE genes (Figure E5)."
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"Notably, a significant number of cells in the early stages of apoptosis were observed in both vector and TNFAIP3 knockdown group, likely due to the effect of lentiviral infection on EC109 growth.Transgenic oeTNFAIP3 KYSE150 were generated by lentiviral infection to overexpress TNFAIP3, and subsequently cultured as oeTNFAIP3 KYSE150‐TRCs (Figures 6D and S6J)."
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"MHV infection caused increased Tnfaip3 gene induction in IRF7 KO cells (Fig. 9A, EV)."
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"PRRSV infection upregulates the expression of negative immune regulators including NF-k B inhibitors (NFKBIA, NFKBID, NFKBIZ, and TNFAIP3) and T-cell exhaustion markers (programmed death ligand-1 [PD-L1], PD-L2, interleukin-10 [IL-10], IDO1, and transforming growth factor b2 [TGFB2]) in PAMs to modulate the host’s immune response [7]."
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"EPs 7630 treatment post-SARS-CoV-2 infection resulted in a significant reduction of IL1B gene expression and strong upregulation of anti-inflammatory TNFAIP3."
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