IndraLab

Statements

USP6 activates pathogenesis. 2 / 2
| 2
reach
"However, TRE17 over expression contribute to tumor pathogenesis [4]."
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reach
"Although this fusion is expected to lead to the high expression of USP6 or be the main mechanism of USP6 gene-mediated tumor pathogenesis, the high percentage of COL1A1 as a fusion partner in these tumor subtypes still likely has a potential association with the osteoid formation on the histology, but further research is needed."
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