IndraLab

Statements

USP22 inhibits Death. 5 / 5
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"A recent study shows that the expression of USP22 protein, a kind of deubiquitinase, can inhibit ferroptotic cardiomyocyte death through the SIRT1/p53/SLC7A11 axis and further protect against myocardial ischemia/reperfusion injury [64]."
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"Overexpression of USP22 could inhibit ferroptotic cardiomyocyte death to protect against IRI (102)."
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"Ma et al. found that ubiquitin-specific peptidase 22 (USP22) inhibits cardiomyocyte death induced by ferroptosis in myocardial I/R injury via the SIRT1/p53/SLC7A11 axis, providing a novel therapeutic target for the treatment [83]."
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"Previous studies have shown that USP22 is widely expressed in various tissues and that USP22 gene deletion can lead to the early death of mouse embryos, whereas heterozygous embryos display stunted gr[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"According to a recent study, USP22 may prevent cardiac ischemia‒reperfusion damage by preventing cardiomyocyte death (Ma S et al., 2020)."
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