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This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

AKT activates USP15. 1 / 1

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"Taken together, our results demonstrate that TGF-beta signaling can regulate p53 stability through upregulation of USP15 translation mediated by the PI3K and AKT pathway and partly elucidate why TGF-beta can suppress cancer progression in early stages when cells express wild-type p53."

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Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...