IndraLab

Statements

LINC00665 activates UCHL3. 3 / 3
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"Collectively, the aforementioned results revealed that LINC00665 functioned as a sponge of miR-582-5p and thus up-regulated UCHL3."
35918714
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"To explore the up-restream mechanism of the potential UCHL3/AhR/PD-L1 axis, we conducted bioinformatics analysis and predicted that LINC00665 may sponge microRNA (miR)-582-5p to mediate UCHL3."
35918714
reach
"Overexpression of LINC00665 and silencing of miR-582-5p enhanced the resistance of NSCLC cells to radiotherapy by upregulating UCHL3 and PD-L1 and stabilizing AhR to promote immune escape (113)."
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