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A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

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INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

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USP17L2 activates NFE2L2. 4 / 4

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"Importantly, ectopic expression of DUB3 caused NRF2 dependent chemotherapy resistance in colon cancer cell lines."

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"Further experiments demonstrated that DUB3 promoted NRF2 stability and transcriptional activity by decreasing the K48 linked ubiquitination of NRF2."

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"DUB3 promotes Nrf2 stability and transcriptional activity by decreasing the K48 linked ubiquitination of Nrf2."

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"Moreover, these authors demonstrated that ectopic expression of DUB3 caused Nrf2 dependent chemotherapy resistance in colon cancer cell lines [XREF_BIBR]."

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INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

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Statements

USP17L2 activates NFE2L2. 4 / 4

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