IndraLab

Statements


| 4

eidos
"Functionally , the loss of Prp8 may specifically inhibit cell viability and metastasis and activated EMT and PI3K / Akt in HCC cells ."

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"Upregulation of Prp8 promoted cell viability and migration in a human hepatic astrocyte line ."

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"The upregulation of Prp8 promoted cell viability , metastasis and the activity of the PI3K / Akt pathway in hepatic astrocytes cells and HCC cells ."

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"Upregulation of Prp8 promoted cell viability and migration in HCC cells ."