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"BmKDfsin5 (1 μM) was found to inhibit the potassium currents mediated by Kv1.1, Kv1.2, Kv1.3, IK, SK3 and hERG channels by approximately 8.7%, 10.2%, 9.0%, 9.1%, 46.3% and 16.9%, respectively ( Fig. 5[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"Due to its TCR induced expression and previously described role in T cell function XREF_BIBR, we hypothesized that enforced expression of Kcna3 may increase potassium efflux with a resultant increase in intratumoural T cell effector function."

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"They proposed that this channel acts in concert with potassium current mediated by Kv1.3 channels, in rat microglia cells activated by lipopolysaccharide (LPS)."

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"One micromolar BmKDfsin3 inhibited the potassium ion currents mediated by Kv1.1, Kv1.2, Kv1.3, IK, SK3 and hERG channels by approximately 57.0%, 27.5, 84.3%, 15.1%, 87.5% and 30.4%, respectively ( Fig[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"

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"Overexpression of the potassium channel Kv1.3 increases potassium efflux from tumor-specific T cells, thereby decreasing intracellular potassium ions, enhancing tumor clearance and survival in mice carrying melanoma, reducing tumor growth, and making T cells more effective in anticancer effects (111)."

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"The Delayed Rectifier Potassium Current Is Driven by Kv1.3 in OBCs of the Degenerated FVB rd1 Retina."