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Mutated USP42 activates rich domain. 1 / 1

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"Expression of USP42 mutants targeting the linker domain and the C-terminal lysine rich domain (XREF_FIG B) showed that although the DUB-inactive USP42 C120A mutant retained the ability to bind histone H2B, this interaction was reduced with the DeltaKK mutant and lost with the Deltalinker mutant (XREF_FIG C)."

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IndraLab

Statements

Mutated USP42 activates rich domain. 1 / 1

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