IndraLab

Statements

STAT3 binds EIF3F. 7 / 7
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"Therefore, our findings suggest that EIF3F could be considered as a novel effector of cell migration and metastasis, and its molecular link with STAT3 fosters the need to pursue investigations on the role of the EIF3F–STAT3 signaling in cancer."
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"The discovery of a role for EIF3F–STAT3 interaction in the genetic control of cell migration and metastasis in human lung adenocarcinoma could lead to the development of diagnosis and therapeutic strategies."
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"XREF_BIBR Esteves et al XREF_BIBR reported that eIF3f interacted with STAT3 and increased snail2 expression, thereby promoting lung cancer metastasis."
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"Accordingly, immunoprecipitation (pull-down) analyses revealed the protein–protein molecular interaction between EIF3F and STAT3 (Supplementary information Fig. xref )."
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"Therefore, our findings identify a novel EIF3F–STAT3 axis involved in the promotion of metastasis."
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"EIF3f knockdown in A549 cells was shown to inhibit cell proliferation and induce apoptosis. xref Esteves et al xref reported that eIF3f interacted with STAT3 and increased snail2 expression, thereby promoting lung cancer metastasis."
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"The interaction between EIF3F and signal transducer and activator of transcription 3 (STAT3) controlled the EIF3F-mediated increase in Snail2 expression and cellular invasion, which were specifically abrogated using the STAT3 inhibitor Nifuroxazide or knockdown approaches."
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