IndraLab

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ATG5 activates mutated USP36. 1 / 1

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"However, the inability of Atg5 knockdown to suppress the USP36 mutant phenotype, as well as the accumulation of both GFP-Atg8a and p62 in USP36 mutant cells, suggests a defect in autophagic flux rather than a defect in the formation of autophagosomes."

23873149

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IndraLab

Statements

ATG5 activates mutated USP36. 1 / 1

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