IndraLab
Statements
Piceatannol inhibits MYD88. 2 / 2
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2
reach
"Indeed, we found that overexpression of myeloid differentiation primary response gene 88 (MyD88), an adaptor molecule that drives TLR signaling, induced IRAK1 expression and that piceatannol, a Syk inhibitor, successfully suppressed the MyD88 dependent upregulation of IRAK1 under LPS treatment conditions."