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OTUD1 inhibits Mice. 2 / 2
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"Our group has recently found that cardiomyocyte OTUD1 promoted angiotensin II‐ and TAC‐driven cardiac dysfunction, hypertrophy and fibrosis, while the role of OTUD1 was evaluated using the whole‐body OTUD1 knockout mice but not the myocardial‐specific OTUD1 knockout mice or BMT."
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"Additionally, OTUD1 and FGL1 depletion significantly restored TNFα-reduced anticancer immunity and prolonged the OS of mice stimulated with TNFα (Fig. 6C, D, S5D), consistent with the above in vitro findings."
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