IndraLab

Statements

Infections activates USP18. 8 / 8
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"The Fig. 1B showed that DENV infection induced the accumulation of USP18 in mitochondria that was dramatically reduced by siUSP18 treatment."
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"Overall, USP18, stimulated by DENV-2 infection, is a critical host factor exploited by DENV-2 to confer IFN-α antagonism [103]."
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"DENV infection induced USP18 which regulated DENV replication through in part IFN-independent manner."
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"Usp18 , Usp49 ) were induced by MTH-68/H infection."
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"This suggests that increased USP18 caused by IAV infection promotes the induction of innate immune responses while promoting IAV replication.IAV can induce host cell apoptosis through an endogenous/mi[MISSING/INVALID CREDENTIALS: limited to 200 char for Elsevier]"
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"In our recent studies, we demonstrate that HIV-1 infection induces USP18, which dramatically enhances HIV-1 replication by abrogating the antiviral function of p21."
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"In a recent study, Ye et al. (2021) showed that USP18 induced by DENV-2 infection is a critical host factor used by DENV-2 to antagonize IFN-α production."
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"We found that HIV-1 infection could induce the USP18 protein in myeloid cell lines."
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