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"Besides, we found that the expression of DHRS2 in the primary tumor was lower than in normal samples in the TCGA-LUAD database (Fig. 3E), while the expression of STAMBPL1 in the primary tumor was higher than in normal samples (Fig. 3F), so we carried out the hypothesis that STAMBPL1 promotes the progression of lung adenocarcinoma by inhibiting DHRS2 expression."