Other Services

This Service

A database built with INDRA combining content from numerous readers and databases. This page allows you to curate the loaded statements. For more information please see the manual.

This Project

INDRA is developed by indralabs, a part of the Harvard Medical School Laboratory of Systems Pharmacology.

This project, indra_db, is also developed by the indralab team. For more information on how we procure our sources, you can go here. This work was funded by ARO grant W911NF‐15‐1‐0544 under the DARPA Communicating with Computers program.

IndraLab

Statements

HTT inhibits ATXN3. 3 / 3

| 3

➶

reach

"Our data indicate that the N-terminal fragment of polyQ-expanded (PQE) Atx7 or Htt can coaggregate with and sequester AR and Atx3 into insoluble aggregates or inclusions through their respective polyQ tracts."

37171184

➶

reach

"PQE Atx7 and Htt reduce the soluble fraction of endogenous Atx3 but increase its insoluble aggregates."

33837238

➶

reach

"PQE Atx7 and Htt promote proteasomal degradation of intracellular Atx3."

33837238

Our Sources:

INDRA allows us to combine the results from a multitude of sources. In particular, the INDRA Database draws on the following...

Other Services

This Service

This Project

IndraLab

Statements

HTT inhibits ATXN3. 3 / 3

Our Sources:

Databases

Reading